CHILDHOOD DEPRESSION

AND LEARNING DISABILITIES

  Renato COCCHI, neurologist and medical psychologist  

Italian translation / testo in italiano  

Summary    

When neurological brain lesions are not evident, or where not found out by current means of investigation, although highly possible as it happens in some forms of mental retardation, and when visual or auditory deficits have been excluded, as well as difficulties in social-cultural integration, learning difficulties in childhood must be supposed to be caused by some form of childhood depression.

Nowadays depression is known to impair cognitive mechanisms such as attention, concentration, memory, thought mobility and so on, and by this way it can decrease learning skills.  A current childhood depression may have remote, somatic (mainly pre-, peri- and neonatal troubles) or recent, somatic or psychological, origin, and often shows a share of secondary psychological-relational depression, because of the first one.  

Only the understanding of these variables can allow remedial intervention less likely to be temporary or incomplete.  

Key words: Learning difficulties; school age; childhood depression; somatic causes; psychological causes.  

Mental retardation

World Congresses on stress and other congresses Symptoms

Home Page // Pagina iniziale

  

As for children with school learning difficulties and our argument, we  have to exclude subjects with burden learning troubles when their brain is evidently damaged as for mental deficiency (MD) in cerebral palsied, in epileptics, or in carriers of chromosomal or genetic anomalies, without any remedial.   

The same we have to say for found out or highly suggested lesions following a pathological history and a current severe, intellectual and social deficit such as in isolated mental deficiency, which, by definition, always presupposes  a previous lesion, even if we are unable to detect it. Nevertheless, the frequency of learning problems in the obligatory school, in an apparently healthy child, can have recent or remote origin.  In this last case, they could not have manifested themselves the preschool years (kindergarten, or, in Italy, "scuola materna"), even because the learning aspects are less formal and less pressing, in that school type.  We should therefore skip all that forms when have been, or have to presuppose a lesion that  has interested brain cognitive areas, with heavy repercussions on the learning, ie. The whole field of the mental deficiency.  

So, when we find learning disabilities that manifest themselves in children lacking any brain lesion, who attend obligatory school, we are facing what in past we preferred to term it  as "false mental deficiency" (FMD) [1].

From a cognitive point of view the FMD has its main feature in the contrast between poor school learning and normal practical performances, (a fact more remarkable in after school career). The IQ of these children is higher than 70 ( the range superior limit of light mental deficiency) and smaller than 90 (the inferior range limit of intellectual normality).  

In fact, what appeared  was only an individual poor answer, apparent and/or temporary, to the tools of intellectual evaluation, by considering the evaluation of the school learning as an intelligence test with long times of application.  Now, the learning disability, in such cases, can be caused by extrinsic factors (environmental: out or in the school itself) or intrinsic ones ( physical and/or psychological factors), which do not correspond however to a brain lesion.  

While in the MD there is the brain damage that inhibits the development of  intellectual abilities, in the FMD, according to this optics, the brain development is normal, but it does not work well, in certain contexts, because  bad running of altered, but not destroyed, structures [2].  

Currently, the evolution of knowledge in this field has brought to a widening idea of FMD, being this last even  present in subjects with MD.  They, combining both conditions, MD + FMD, do fewer intellectual performances  than what they really can [2].

Causes of learning disabilities in the obligatory school.

By searching  possible causes  of the learning disability, we shall start from the common final datum.  When learning disability appears, nearly always it  is  present even a secondary psychological depression. In its turn, it more contributes to impair  the necessary cognitive mechanisms ( attention, concentration, memory,  thought motility,  the use of second or third degree integrating mechanisms, etc.), already ill-working  following the primary cause.  

The secondary depression comes from the acquired awareness of own learning difficulty, and, throughout this way, it  increases a cognitive situation already impaired. Besides  in the FMD, it is ( nearly ) always present even in the MD.  

Anyhow, this share is the easier to treat with simple support (like encouragement,  pointing up the successes rather than the errors; If possible and in needs, a more efficacious method of studying may be proposed), with noticeable results both  in pure FMD and in FMD added to MD [3].

 In the MD, which originates from brain damage by definition, any IQ improvement cannot  come from  decreasing  of the coexistent share of FMD [2].

 If instead we are want to investigate the FMD primary causes, it is custom distinguish them as extrinsic and intrinsic factors.  

Extrinsic factors:  

a - culture linked factors.

I am referable to a certain reduction  of  intellectual performances given by subjects belonging to another cultural contest. Every individual assimilates the culture of his/her origin group and consequently, if  the child has to adapt to a new cultural context, he/she can meet some difficulties  in adapting him/herself  to new schemes.  It is very probable that this type of factor will have strong impact in  schooling of sons of immigrants from  extra-European countries who entered  Italy in bulk in recent times.

 b - economic and family factors. They are:  long absences of the parents;  the need  mother's working (for which the children are, for part of the day, left to themselves); the disagreement among the parents; educational errors before the school age;  requested or excessive expectations by the parents, as for the scholastic success of the child; to be the last born in a large family [2].

c - factors linked to school  structures and dynamics.  They can act either trough somatic pathways (with an increase of the physical fatigue) and through psychological-relational pathways ( for disturbed relationship with the teacher or with the classmates).  I shall stay on only the list of them: Distance from the school, class with many pupils, insufficient or unsuitable classrooms, a double or triple sharing of the same classroom, class made up of different age groups,  quick turning of the teachers of  the same class, disturbed personality of the teacher, isolation among the peers' group, etc. [10].  

All the extrinsic factors, at least took a for a, have to be considered more properly as concurrent / co-operating causes, able to express  their negative influence on a frail child [4].

 If all these possible causes were enough, as one for itself, we should have that every child, when a sufferer from at least  one of them, should  go to have learning problems. A fact  that  common evidence denies.

Intrinsic factors:  

d - physical conditions: Sensory troubles, and of the language, motor deficits, convulsive fits, long-time use of anticonvulsant drugs that inhibit learning abilities, weakening illnesses, poor diet [9].

 Moreover all the states of not optimality happened in pre-  peri- and neonatal age, which apparently did not resulted in some immediate troubles. Only then they showed to have  impaired brain functional areas recruited for systematic school learning  [9].

To these events we have to add  many other postnatal ones, among which cranial traumas and  followings of meningitis or encephalitis, both overcome  without any residual objective neurological troubles.  The same we can say for whooping cough,  and  long therapies with antibiotics, whose depressant effects on the brain cannot be ignored further on.

 In after all,  we are dealing with causes able to produce a childhood depression of somatic origin. From a psychic point of view, besides  learning troubles, it can manifest itself  with emotional inhibition or  the so-called temper reactivity [9].  

In this field we have also to remember:

- the asthenia, with the child that accuses serious fatigue and does not have any endurance;

-  over emotionality  on a genetic base, with distraction easiness and serious insecurity;

- the syndrome from the deficit of the attention with or without hyperactivity;

-  or, finally, a simple delay in intellectual maturation. The child develops slowly and shows a delay when compared with  age peers. 

 His psychic state  can be summarised in a psychological-relational childhood depression, which may even manifest itself with inhibition or the so-called temper reactivity.   

 

The depression as an unifying moment.

We do not intend as depression only the mood decreasing, which, mainly in the infancy, can even be lacking.  In  neuro-pathological terms,  we mean a reduced working of areas, or structures or neuronal mechanisms. Then, with this term, we may point out a unifying element that permits to understand all the cognitive troubles on a dysfunctional  base, among which even those due to a childhood depression, in a psychological-relational sense.  

Excluding the secondary psychological depression, from acquired awareness of own school difficulties, we however  have to remember these four facts:

- the cognitive trouble seems joining only the 2/3 of  manifest childhood depressions;

-  the same can be the only evident symptom of a remote or recent childhood  somatic depression; - the current depressive state (in widen sense) can be fruit of many causal factors. Each of them by alone is insufficient to produce that depressive condition, instead  due to their summation;

- the presence of a psychological component doesn't have immediately to drive to conclude that the learning trouble comes ONLY from psychological factors.   

Besides being this psychological  component  of primary origin ( a case where the diagnosis is exact ), it can even be only concomitant with a somatic cause.

Finally it can be  only a secondary reactive component, due to the awareness of own learning disabilities. The last one can be found even in mentally retarded Ss where it elicits its own share of reduced intellectual performances.

 

Conclusions.

Learning disabilities in a child without any evident or presupposed neurological trouble, and without sensory troubles can have their explanation on a neural dysfunction on a depressive base, in a wider sense.

This dysfunction can hinder the cognitive mechanisms that permit learning. To know all the possible somatic or psychological causes, either in the past or in recent time, either primary or secondary, often present together in a varius number, forces to an accurate anamnestic investigation, and ponts up the way for a more targeted and complete intervention.  

 

References.

[1] Cocchi R., Tornati A.: Alcuni rilievi sul concetto e sul fenomeno di pseudoinsufficienza mentale. Rass. Studi Psichiat. 1975, 64: 546-556.  

[2] Cocchi R.: La pseudoinsufficienza mentale. In: Strutture e dinamiche neuropsicopatologiche in eta` evolutiva. Montefeltro, Urbino 1985.  

  [3] Buckley S., Emslie M., Haslegrave G., LePrevost P.: The development of language and reading skills in children with Down's syndrome. Portsmouth Polytechnic, Portsmouth 1986 (trad. it. GISSTIMMAI, San Costanzo 1989).

 [4] Cocchi R.: Il disadattamento scolastico. In: Strutture e dinamiche neuropsicopatologiche in eta` evolutiva. Montefeltro, Urbino 1985.

  [5] Blemont L., Stein Z.A., Witterķ J.T.: Birth order, family size and school failure. Develop. Med. Child Neurol. 1976, 18: 421-430.  

[6] Cocchi R.: Prime esperienze sull'uso di una associazione a bassī dosaggio di amitriptilina + perfenazina nella terapia del disadattamento scolastico. Rass. Studi Psichiat. 1974, 63: 862-870.

  [7] Murray M.E., Little M.: Depression in learning disabled children. J. Psychiat. Treat. Eval. 1981, 3: 193-196.  

 [8] Brumback R.A., Staton R.D.: Learning disability and childhood depression. Am. J. Orthopsychiat. 1983, 53: 269-281.  

[9] Cocchi R.: Le depressioni infantili. In: Strutture e dinamichå neuropsicopatologiche in eta` evolutiva. Montefeltro, Urbino 1985.  

[10] Cocchi R., Tornati A., Cocchi Cercolani P.: Depressione e disadattamento nella scuola dell'obbligo. Tentativo di analisi intedisciplinare. Note Riv. Psichiat. 1973, 66: 321-360.  

 

Paper presented during the IX World Congress of the IASSMD. Brisbane / Broadbench,  1992.

Printed on It. J. Intellect.
Impair. 1992, 5: 137-142.

 

Author's address : dr Renato COCCHI, via via Rabbeno, 3

42100 Reggio Emilia (Italy)

renatococchi@libero.it

Testo in italiano

Mental retardation

World Congresses and other Congresses

Home Page // Pagina iniziale